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As with any disease or painful condition, it is essential to understand the etiology of tooth sensitivity to plan for effectively managing client discomfort and preventing subsequent painful episodes. However, hypersensitivity is a confounding condition because its development and expression is influenced by modifying and predisposing factors as well as by individual susceptibility. Additionally, an individuals psychogenic pain response impacts the perception of the severity of the condition and the benefits of treatment.
Hypersensitivity has been referred to as an enigma1,2 for a variety of reasons, including difficulty in determining the etiology, the numerous treatment approaches, variability in pain relief, and because invivo objective clinical trials are difficult to conduct. A prerequisite for the occurrence of hypersensitivity is gingival recession and subsequent dentin exposure as a result of loss of cementum or enamel. Reasons for cementum and enamel loss are often difficult to determine.
There is no consensus in the literature concerning terminology used to describe the condition. The terms dentin hypersensitivity and tooth hypersensitivity or hypersensitivity have been questioned because the pulp response that is elicited by the stimulation of exposed dentin would be expected as a normal rather than a hyper-reaction.3 The terms dentinal, dentin, or tooth sensitivity frequently have been used. The condition has also been referred to as cervical dentin sensitivity4,5 and as cervical tooth sensitivity,6 adding a locationbased descriptor to differentiate it from other types of tooth pain.
For this continuing education course, the terms dentin hypersensitivity or hypersensitivity will be used. |
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ŠADHA
2003
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